Luring BRCA1 to the scene of the crime.

Cancer cell ·第 23 卷 ·第 5 期 ·2013-07-22

Baer Richard

摘要

To preserve genome stability, BRCA1 must be recruited to sites of DNA damage, where BRCA1 facilitates repair of double-strand DNA breaks (DSBs). In this issue of Cancer Cell, Li and Yu report that BRCA1 recruitment involves a novel interaction between its partner protein BARD1 and poly(ADP-ribose) chains at the DSB.

文献信息

期刊

Cancer cell

期刊简称

Cancer Cell

ISSN

1535-6108

发表日期

2013-07-22

收录日期

2013-05-17

更新日期

2016-11-25

语言

英语

国家/地区

United States

NLM ID

101130617

外部链接

PubMed 原文查看期刊详情

分析服务

Luring BRCA1 to the scene of the crime.

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